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Vascular access malfunction: towards a more genecentric view.

ΤίτλοςVascular access malfunction: towards a more genecentric view.
Publication TypeJournal Article
Year of Publication2013
AuthorsKirmizis, D., Chatzidimitriou D., Chatzopoulou F., Tzimagiorgis G., Malisiovas N., & Papagianni A.
JournalClin Nephrol
Volume80
Issue5
Pagination370-6
Date Published2013 Nov
ISSN0301-0430
Λέξεις κλειδιάAged, Arteriovenous Shunt, Surgical, Factor V, Heme Oxygenase-1, Humans, Interleukin-10, Male, Methylenetetrahydrofolate Reductase (NADPH2), Middle Aged, Peptidyl-Dipeptidase A, Renal Dialysis, Transforming Growth Factor beta1
Abstract

A well-functioning vascular access is the cornerstone for an optimal hemodialysis treatment and an issue of major importance for the outcome of patients on chronic hemodialysis. Over the last few years reports supporting the aspect that mechanisms involved in vascular access malfunction are genetically controlled have been published. Triggered by two cases reported herein, we present a comprehensive review of the literature on an evolving field of particular significance to patients on hemodialysis.

DOI10.5414/CN107071
Alternate JournalClin. Nephrol.
PubMed ID22541675

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